Journal of immunology (Baltimore, Md. : 1950)
Distinct T cell developmental consequences in humans and mice expressing identical mutations in the DLAARN motif of ZAP-70.
Cutting edge: tyrosine-independent transmission of inhibitory signals by CTLA-4.
Natural killing of xenogeneic cells mediated by the mouse Ly-49D receptor.
Pre-T cell receptor signals are responsible for the down-regulation of Syk protein tyrosine kinase expression.
Identification of tyrosine phosphorylation sites in the CD28 cytoplasmic domain and their role in the costimulation of Jurkat T cells.
Nuclear factor of activated T cells and AP-1 are insufficient for IL-2 promoter activation: requirement for CD28 up-regulation of RE/AP.
Phosphatidylinositol 3-kinase is required for CD28 but not CD3 regulation of the TEC family tyrosine kinase EMT/ITK/TSK: functional and physical interaction of EMT with phosphatidylinositol 3-kinase.
Mutational analysis of CD28-mediated costimulation of Jun-N-terminal kinase and IL-2 production.
Differential requirements for ZAP-70 in TCR signaling and T cell development.
The alpha2 domain of H-2Dd restricts the allelic specificity of the murine NK cell inhibitory receptor Ly-49A.
